Frederick J. Suchy, Ronald J. Sokol, William F. Balistreri's Liver Disease in Children PDF

By Frederick J. Suchy, Ronald J. Sokol, William F. Balistreri

ISBN-10: 0511286457

ISBN-13: 9780511286452

ISBN-10: 0521856574

ISBN-13: 9780521856577

Thoroughly revised new version of the most suitable reference on pediatric liver affliction. Liver disorder in teenagers, third version presents authoritative insurance of each point of liver illness affecting babies, childrens, and kids. Chapters are written through foreign specialists and tackle the original pathophysiology, manifestations, and administration of those issues within the pediatric inhabitants. The 3rd variation has been completely up to date and lines new contributions on liver improvement, cholestatic and autoimmune problems, fatty liver disorder, and inborn blunders of metabolism. an important source for all physicians interested by the care of kids with liver ailment.

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Amino Acid Metabolism Amino acids are an important source of energy for the fetus [8]. Amino acids as a metabolic fuel provide an amount of energy for the fetus equivalent to that provided by glucose. Even essential amino acids are oxidized for energy in the fetus. In animal studies, amino acids account for approximately one third of fetal carbon uptake and over 40% of fetal energy requirements. In rodent studies, the uptake of glutamine, alanine, and lysine by the liver is much greater than their incorporation into protein.

Pancreatic tumoral cell line AR42J: an amphicrine model. Am J Physiol 1994;266:G963–71. ■ 13 117. Mashima H, Shibata H, Mine T, Kojima I. Formation of insulinproducing cells from pancreatic acinar AR42J cells by hepatocyte growth factor. Endocrinology 1996;137:3969–76. 118. Shen CN, Slack JM, Tosh D. Molecular basis of transdifferentiation of pancreas to liver. Nat Cell Biol 2000;2:879–87. 119. Lee BC, Hendricks JD, Bailey GS. Metaplastic pancreatic cells in liver tumors induced by diethylnitrosamine.

2) [34]. There have been many recent advances in understanding of the mechanisms that regulate the expression of genes encoding the enzymes and transporters involved in drug metabolism. The xenobiotic nuclear receptors (NRs), the pregnane X receptor (PXR, also known as the steroid and xenobiotic receptor, or SXR), the constitutive androstane receptor (CAR), and the aryl hydrocarbon receptor (AhR) coordinately induce genes involved in the three phases of xenobiotic metabolism, including oxidative metabolism, conjugation, and transport [35,36].

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Liver Disease in Children by Frederick J. Suchy, Ronald J. Sokol, William F. Balistreri

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