Download e-book for kindle: Evidence-based Gastroenterology and Hepatology, Second by

ISBN-10: 0470987022

ISBN-13: 9780470987025

ISBN-10: 072791751X

ISBN-13: 9780727917515

Conventional textbooks during this box have emphasised the elemental sciences of pathology, biochemistry and body structure. Evidence-based Gastroenterology and Hepatology covers the entire significant illnesses of the gastrointestinal tract and liver, using scientific epidemiology to give the most powerful and most modern facts for interventions.

This moment variation is edited and written by way of major gastroenterologists from around the globe, each one bankruptcy summarizes the proof in order that larger expert judgements may be made approximately which remedies to provide to patients.

It offers working towards Gastroenterologists and Surgeons with transparent information about the prognosis and remedy of pancreatic ailments, giving transparent facts and experience-based fabric that's instantly suitable to medical practice.

Also features a record of urged studying on the finish of every chapter.

Take a glance at modern info at www.evidbasedgastro.com

Content:
Chapter 1 creation (pages 1–11): John WD McDonald, Brian G Feagan and Andrew ok Burroughs
Chapter 2 Gastroesophageal Reflux illness (pages 13–54): Naoki Chiba
Chapter three Barrett's Esophagus (pages 55–68): Carlo A Fallone, Marc Bradette and Naoki Chiba
Chapter four Esophageal Motility problems: Achalasia and Spastic Motor problems (pages 69–81): Marcelo F Vela and Joel E Richter
Chapter five Ulcer sickness and Helicobacter Pylori (pages 83–116): Naoki Chiba
Chapter 6 Non?Steroidal Anti?Inflammatory Drug?Induced Gastroduodenal Toxicity (pages 117–138): Alaa Rostom, Andreas Maetzel, Peter Tugwell and George Wells
Chapter 7 Non?Variceal Gastrointestinal Hemorrhage (pages 139–159): Nicholas Church and Kelvin Palmer
Chapter eight practical Dyspepsia (pages 161–168): Sander JO Veldhuyzen van Zanten
Chapter nine Celiac affliction (pages 169–178): James Gregor and Diamond Sherin Alidina
Chapter 10 Crohn's illness (pages 179–195): Brian G Feagan and John WD McDonald
Chapter eleven Ulcerative Colitis (pages 197–210): Derek P Jewell and Lloyd R Sutherland
Chapter 12 Pouchitis After Restorative Proctocolectomy (pages 211–219): William J Sandborn
Chapter thirteen Microscopic and Collagenous Colitis (pages 221–229): Robert Lofberg
Chapter 14 Metabolic Bone sickness in Gastrointestinal problems (pages 231–246): Ann Cranney, Catherine Dube, Alaa Rostom, Peter Tugwell and George Wells
Chapter 15 Colorectal melanoma in Ulcerative Colitis: Surveillance (pages 247–253): Bret A Lashner and Alastair JM Watson
Chapter sixteen Colorectal melanoma: inhabitants Screening and Surveillance (pages 255–263): Bernard Levin
Chapter 17 Irritable Bowel Syndrome (pages 265–283): Albena Halpert and Douglas A Drossman
Chapter 18 Clostridium Difficile affliction (pages 285–301): Lynne V McFarland and Christina M Surawicz
Chapter 19 Ogilvie's Syndrome (pages 303–309): Michael D Saunders and Michael B Kimmey
Chapter 20 Gallstone disorder (pages 311–320): Calvin HL legislation, Dana McKay and Ved R Tandan
Chapter 21 Acute Pancreatitis (pages 321–339): Jonathon Springer and Hillary Steinhart
Chapter 22 weight problems (pages 341–357): Jarol Knowles
Chapter 23 Hepatitis C (pages 359–366): Patrick Marcellin
Chapter 24 Hepatitis B (pages 367–381): Piero Almasio, Calogero Camma, Vito Di Marco and Antonio Craxi
Chapter 25 Alcoholic Liver sickness (pages 383–391): Philippe Mathurin and Thierry Poynard
Chapter 26 Non?Alcoholic Fatty Liver disorder (pages 393–403): Chris P Day
Chapter 27 Hemochromatosis and Wilson affliction (pages 405–413): Gary Jeffrey and Paul C Adams
Chapter 28 basic Biliary Cirrhosis (pages 415–426): Jenny Heathcote
Chapter 29 Autoimmune Hepatitis (pages 427–434): Michael Peter Manns and Andreas Schuler
Chapter 30 basic Sclerosing Cholangitis (pages 435–451): Roger Chapman and Sue Cullen
Chapter 31 Portal Hypertensive Bleeding (pages 453–485): John Goulis and Andrew okay Burroughs
Chapter 32 Ascites, Hepatorenal Syndrome, and Spontaneous Bacterial Peritonitis (pages 487–503): Pere Gines, Vicente Arroyo and Juan Rodes
Chapter 33 Hepatic Encephalopathy (pages 505–515): Peter Ferenci and Christian Muller
Chapter 34 Hepatocellular Carcinoma (pages 517–525): Massimo Colombo
Chapter 35 Fulminant Hepatic Failure (pages 527–543): Nick Murphy and Julia Wendon
Chapter 36 Liver Transplantation: Prevention and therapy of Rejection (pages 545–571): Laura Cecilioni, Lucy Dagher and Andrew Burroughs
Chapter 37 Liver Transplantation: Prevention and remedy of an infection (pages 573–586): Nancy Rolando and Jim J Wade
Chapter 38 administration of Hepatitis B and C After Liver Transplantation (pages 587–601): George V Papatheodoridis and Rosangela Teixeira

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Extra resources for Evidence-based Gastroenterology and Hepatology, Second Edition

Example text

261 Ald It is interesting that sucralfate, which does not lower acid output, reduce esophageal acid exposure or improve esophageal transit time262 has any efficacy given our understanding of the pathophysiology of this condition. The adverse effect of constipation, the need for four times daily dosing and the modest observed benefit make sucralfate an unattractive choice for most patients. 14,19,228,230,263–279 Ald In the meta-analysis,11 the rate of healing, expressed as “percent healed per week”, was significantly superior with PPI therapy compared with H2-RA, particularly early in the course of treatment (weekly healing rate in first 2 weeks: PPI 32%, H2-RA 15%).

48 The combination of the presence of grade 2 or 3 symptoms on the standardized questionnaire and endoscopic esophagitis, predicted increased acid exposure on 24-hour intraesophageal pH monitoring with a specificity of 97% and a positive predictive value of 98%. 1 Acid exposure of the distal part of the esophagus during eight 3-hour periods expressed as median % time spent with pH < 4 in 190 patients with different degrees of heartburn and acid regurgitation and 50 asymptomatic endoscopically normal subjects.

The negative predictive value of a low SI is useful. A limitation of the SI is that it does not take into account the reflux episodes that were symptom free. 148 This method correlates pH data 22 during both symptomatic and asymptomatic reflux episodes and requires further validation. Therapeutic trial of acid suppression as a diagnostic test All of the diagnostic tests described above are cumbersome or invasive and detect different features of reflux. PPIs such as omeprazole are the most effective intervention for all grades of esophagitis and for treatment of symptoms such as heartburn.

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